Not quite true. You are correct about the sino-atrial node having a major role: it's a collection of cells that can create a rhythmical pacemaker potential spontaneously, and is a big influence on the heartrate. However, the CNS does also have a huge role in regulating the heart rate, through both parasympathetic (vagus) nerves and sympathetic (from the sympathetic trunk) nerves. The vagus nerve acts on the SA node, releasing a neurotransmitter (acetylcholine) that slows the generation of action potentials by the node, overall slowing the heartbeat. (Side note: atropine, a drug given sometimes during a cardiac arrest, blocks these neurotransmitters from binding the heart cells, thereby preventing the Vagus nerve from slowing the heartrate.) The sympathetic nerves coming from T2-T4 of the spine are more diffuse in their effect, hitting the entire heart and releasing the neurotransmitter norepinephrine. It can increase the heartrate, force of contraction and speed of electrical conduction within the heart. (This is why beta-blockers can be used to slow down the heartrate, by preventing norepinephrine from binding to the heart.)